The Classical Complement Cascade Mediates CNS Synapse Elimination

As a Potential Therapeutic Target, C1q Induces Synapse Loss Via Inflammasome-activating Apoptotic and Mitochondria Impairment Mechanisms in Alzheimer's Disease

Full article: The case for complement component 5 as a target in neurodegenerative disease

Biomedicines, Free Full-Text

C1q: the perfect complement for a synaptic feast?

Glia, Neurobiology Journal

Frontiers Complement Activation in the Central Nervous System: A Biophysical Model for Immune Dysregulation in the Disease State

Microglial trogocytosis and the complement system regulate axonal pruning in vivo

Complement C3-dependent glutamatergic synapse elimination in the developing hippocampus is region- and synapse-specific

The Classical Complement Cascade Mediates CNS Synapse Elimination: Cell

Figure 2 from The complement system: an unexpected role in synaptic pruning during development and disease.

A new age for (mucosal) NeuroImmunology - Mucosal Immunology

Complement and microglia mediate early synapse loss in Alzheimer mouse models

Mechanisms governing activity-dependent synaptic pruning in the developing mammalian CNS