Modulation of myeloid and T cells in vivo by Bruton's tyrosine kinase inhibitor ibrutinib in patients with metastatic pancreatic ductal adenocarcinoma
Por um escritor misterioso
Last updated 17 outubro 2024
Full article: Immunosuppressive tumor microenvironment modulation by chemotherapies and targeted therapies to enhance immunotherapy effectiveness
T cells infiltrate PDA regardless of GM-CSF expression. A-C) IHC of
Ibrutinib Single-handedly Impacts Immune Environment in Metastatic PDAC
Scientific rationale for integrative and personalised strategies for pancreatic ductal adenocarcinoma management
Vaccines, Free Full-Text
Cancers, Free Full-Text
Sulfonamides as tyrosine kinase modulators – A promising class of anticancer agents - ScienceDirect
The immunological landscape in pancreatic ductal adenocarcinoma and overcoming resistance to immunotherapy - The Lancet Gastroenterology & Hepatology
Therapeutic advances in metastatic pancreatic cancer: a focus on targeted therapies - Anthony Turpin, Cindy Neuzillet, Elise Colle, Nelson Dusetti, Rémy Nicolle, Jérôme Cros, Louis de Mestier, Jean-Baptiste Bachet, Pascal Hammel, 2022
Envisioning the immune system to determine its role in pancreatic ductal adenocarcinoma: Culprit or victim? - ScienceDirect
The recent progress of myeloid‐derived suppressor cell and its targeted therapies in cancers - Ren - 2023 - MedComm - Wiley Online Library
Protein tyrosine kinase inhibitor resistance in malignant tumors: molecular mechanisms and future perspective
M1 Macrophage-Derived Nanovesicles Potentiate the Anticancer Efficacy of Immune Checkpoint Inhibitors
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